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| Registros recuperados : 57 | |
| 47. |  | ZANARDI, O. Z.; NAVA, D. E.; BOTTON, M.; GRUTZMACHER, A. D.; MACHOTA JUNIOR, R.; BISOGNIN, M. Desenvolvimento e reprodução da mosca-do-mediterrâneo em caquizeiro, macieira, pessegueiro e videira. Pesquisa Agropecuária Brasileira, Brasília, DF, v. 46, n. 7, p. 682-688, jun. 2011 Título em inglês: Development and reproduction of the Mediterranean fruit fly in persimmon, apple, peach and grape.| Biblioteca(s): Embrapa Clima Temperado; Embrapa Unidades Centrais; Embrapa Uva e Vinho. |
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| 49. | | MACHOTA JUNIOR, R.; BORTOLI, L. C.; BOTTON, M. Efeito da flubendiamida sobre Anastrepha fraterculus (Wied.) (Diptera: Tephritidae) em laboratório. In: CONGRESSO DE INICIAÇÃO CIENTÍFICA, 19.; ENCONTRO DE PÓS-GRADUAÇÃO, 12.; MOSTRA CIENTÍFICA, 2., 2010, Pelotas. Que futuro queremos: etica, ciência, política: [anais]. Pelotas: UFPEL, 2010. Não paginado. Resumo expandido.| Biblioteca(s): Embrapa Uva e Vinho. |
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| 51. | | ZANARDI, O. Z.; NAVA, D. E.; BORGES FILHO, R. da C.; VERÍSSIMO, M. A. A.; SILVA, S. D. dos A. e; MACHOTA JUNIOR, R. Identificação e caracterização da época de ocorrência dos principais Artrópodes-praga do pinhão-manso no município de Pelotas, RS. In: SIMPÓSIO ESTADUAL DE AGROENERGIA, 3.; REUNIÃO TÉCNICA ANUAL DE AGROENERGIA, 3.; REUNIÃO TÉCNICA DA MANDIOCA, 10.; REUNIÃO TÉCNICA DA BATATA-DOCE, 2., Pelotas, 2010. Anais... Pelotas: Embrapa Clima Temperado, 2010. Organizado por Sérgio Delmar dos Anjos e Silva, Ivan Rodrigues de Almeida e Ana Cláudia Barneche de Oliveira. 1 CD-ROM.| Biblioteca(s): Embrapa Clima Temperado. |
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| 52. | | BORGES, R.; BOTTON, M.; MACHOTA JUNIOR, R.; BOFF, M. I. C.; BORTOLI, L. C.; MAFRA-NETO, A. Persistence of Attract & Kill formulations of Anastrepha Fraterculus: greenhouse and rain-fast experiments. In: INTERNATIONAL SYMPOSIUM ON FRUIT FLIES OF ECONOMIC IMPORTANCE, 8., Valencia, 2010. [Abstracts...] [Valencia]: Universitat Politècnica de Valencia, 2010. p. 293. Resumo.| Biblioteca(s): Embrapa Uva e Vinho. |
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| 53. | | VERISSIMO, M. A. A.; SILVA, S. D. dos A. e; ZANARDI, O. Z.; MACHOTA JUNIOR, R.; NAVA, D. E. Preferência de ataque do ácaro-branco [Polyphagotarsonemus latus (Banks, 1904) (Acari: tarsonemidae)] em genótipos de pinhão-manso, em condições de campo. In: SIMPÓSIO ESTADUAL DE AGROENERGIA, 3.; REUNIÃO TÉCNICA ANUAL DE AGROENERGIA, 3.; REUNIÃO TÉCNICA DA MANDIOCA, 10.; REUNIÃO TÉCNICA DA BATATA-DOCE, 2., Pelotas, 2010. Anais... Pelotas: Embrapa Clima Temperado, 2010. Organizado por Sérgio Delmar dos Anjos e Silva, Ivan Rodrigues de Almeida e Ana Cláudia Barneche de Oliveira. 1 CD-ROM.| Biblioteca(s): Embrapa Clima Temperado. |
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| Registros recuperados : 57 | |
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 | Acesso ao texto completo restrito à biblioteca da Embrapa Pantanal. Para informações adicionais entre em contato com cpap.biblioteca@embrapa.br. |
Registro Completo
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Biblioteca(s): |
Embrapa Pantanal. |
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Data corrente: |
19/03/1997 |
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Data da última atualização: |
31/03/2017 |
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Autoria: |
RIVERA DAVILA, A. M.; SILVA, R. A. M. S.; RAMIREZ, L. |
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Afiliação: |
UFMS. Centro Universitario de Corumba (Corumba, MS); EMBRAPA. Centro de Pesquisa Agropecuaria do Pantanal (Corumba, MS). |
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Título: |
Protection from lethal infection by prior immunization with an homologous Trypanosoma evansi Strain. |
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Ano de publicação: |
1995 |
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Fonte/Imprenta: |
In: CONGRESSO DA SOCIEDADE BRASILEIRA DE PARASITOLOGIA, 14., 1995, Goiania. Resumos... Goiania: [s.n.] 1995. p.325. |
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Idioma: |
Inglês |
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Conteúdo: |
Trypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance of trypomastigotes was successful on 1 day-post-quimiotherapy (d.p.q.) Seventeen d.p.q. rats were re-chanllenge with log. 7.69 trypomastigotes of homologous variant (double dose of first challenge), protection was successful during 49 after first-challenge, when animals were re-challenge with log 8.47 trypamastigotes of homologous variant (1345% more first inocolum) obtained of sucessive syringe passaged in others Wistar rats. Partial protection was observed, prepatent period was of 4 days, and log. 67,7/ml (in 75% of rats) and log. 8.52/ml (in 25% of rats) on the first and 9th day of parasitemia were respectivily registred. Control group parasitemia was log 6.38/ml and log 8.7/ml on first and 5th d.p.i. (died). Possible antigenic variation of strain on the third challenge or overdose of inocolum are postuled. Serum sample were collected during experiment for after evaluation. We verificated protection conferred against homologous T. evansi strain, as demonstred by others authors with T. congolense, T. brucei, T. cruzi and T. evansi. More studies are necessary to determinate which mechanisms are involved in the partial protection. MenosTrypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance ... Mostrar Tudo |
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Palavras-Chave: |
Infeccao letal; Lethal infection. |
|
Thesagro: |
Imunização. |
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Thesaurus NAL: |
immunization; Pantanal; Trypanosoma evansi. |
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Categoria do assunto: |
-- |
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Marc: |
LEADER 03338naa a2200217 a 4500
001 1790947
005 2017-03-31
008 1995 bl --- 0-- u #d
100 1 $aRIVERA DAVILA, A. M.
245 $aProtection from lethal infection by prior immunization with an homologous Trypanosoma evansi Strain.
260 $c1995
520 $aTrypanosoma evansi is a salivarian trypanosome belonging to subgenus Trypanozoon. It is the causative agent of Surra in the Old World and Mal de Caderas in the subtropical area of Argentina and Pantanal, Brazil. Differents methods are used for protection against salivarian-trypanosome infection: a)infection with irradiated trypanosomes, b)infection with trypanosome components and c) infection and quimiotherapy. Only the ultimate is used with successful results for homologous but not heterologous challenges. Survival of acute Trypanosoma cruzi infection has been suggested to be develop in naturally susceptible host and can be enhanced in inherently resistant animals by prior immunization. Highly susceptible C3H/He strain of laboratory mice can be protected froman otherwise lethal infection by prior immunization with an avirulent T.cruzi strain. Enhanced resistance of Balb/c mice to T.congolense infection with 10.3 but not 10.5 of homologous variants was related. Also has been reported protection its conferred for non-surface components antigens in homologous T.evansi challenge. Athimic mice are capable of control T.brucei and T.rhodesiense replication and become resistant to subsequente challenge with homologous parasites. Four males Wistar rats weighting 169.5 g were challenged with log. 7.34 trypomastigotes of T. evansi. Mean parasitemia was log 6.45/ml on the first day-post-infection (d.p.i.) in 100% of rats. All animals were given Berenil (7mg/Kg) on 4th d.p.i. Clearance of trypomastigotes was successful on 1 day-post-quimiotherapy (d.p.q.) Seventeen d.p.q. rats were re-chanllenge with log. 7.69 trypomastigotes of homologous variant (double dose of first challenge), protection was successful during 49 after first-challenge, when animals were re-challenge with log 8.47 trypamastigotes of homologous variant (1345% more first inocolum) obtained of sucessive syringe passaged in others Wistar rats. Partial protection was observed, prepatent period was of 4 days, and log. 67,7/ml (in 75% of rats) and log. 8.52/ml (in 25% of rats) on the first and 9th day of parasitemia were respectivily registred. Control group parasitemia was log 6.38/ml and log 8.7/ml on first and 5th d.p.i. (died). Possible antigenic variation of strain on the third challenge or overdose of inocolum are postuled. Serum sample were collected during experiment for after evaluation. We verificated protection conferred against homologous T. evansi strain, as demonstred by others authors with T. congolense, T. brucei, T. cruzi and T. evansi. More studies are necessary to determinate which mechanisms are involved in the partial protection.
650 $aimmunization
650 $aPantanal
650 $aTrypanosoma evansi
650 $aImunização
653 $aInfeccao letal
653 $aLethal infection
700 1 $aSILVA, R. A. M. S.
700 1 $aRAMIREZ, L.
773 $tIn: CONGRESSO DA SOCIEDADE BRASILEIRA DE PARASITOLOGIA, 14., 1995, Goiania. Resumos... Goiania: [s.n.] 1995. p.325.
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